Download Cancer Cell Lines, Part 1 by J.R. Masters, B. Palsson PDF

By J.R. Masters, B. Palsson

This sequence presents the 1st whole consultant to the tradition of human cells. it's a distinct handbook and reference source for researchers in each element of drugs, biology, expertise and immunobiology utilizing human cells. each bankruptcy is written by way of an the world over acknowledged authority within the box. the 1st 3 volumes within the sequence offer a finished textual content on the tradition of confirmed cellphone traces from all kinds of human melanoma. they supply a easy guide and reference source for each melanoma learn scientist utilizing human melanoma cells.

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Mol Cell Biol. 13:7447–56, 1993. Peverali, F. et al. Oncogene. 12:457–462, 1996. Ucar, K. et al. Cancer Gene Therapy. 2:171–181, 1995. Gross, N. et al. Int J Cancer. 59:141–148, 1994. Lucarelli, E. et al. Submitted, 1998. Neuroblastoma 126. 127. 128. 129. 130. 131. 132. Raschella, G. et al. J Biol Chem. 270:8540–8545, 1995. Judware, R. and Culp, L. Oncogene. 14:1341–1350, 1997. Cohen, P. S. et al. Blood 84:3465, 1994. Beck, D. et al. Blood 86:3132–3138, 1995. Johnson, J. A. M. J Biol Chem 269:18856–63, 1994.

BM =bone marrow; TD = tissue dispersion; C =direct culture; DG = density gradient; LN = lymph node; PB =peripheral blood; NM = nude mouse; unk= unknown, pc =personal communication Cell line 103 104 105 106 107 108 109 110 111 112 113 Table 1 (continued) 28 Thiele 1 2 3 4 5 6 7 8 9 10 11 12 13 14 15 16 17 18 19 20 21 22 23 24 25 + + + ? + unk. + ? 2 POG 1771 POG 1643 POG 1691 RN-GA NBL-S NBL-W NUB-7 Cell line Table 2 (continued) + + + + + + + + + + + ? + + - + + - - + + + 1p alteration 1pdel t(1p) + + + + + + + + + + - + + + - + - - + + + + + + + N - myc amplification HSR DM + + + + + + + + + N - myc single copy Ref (53) (78) (79) (78) -17; der(17) (80) i(17q) " (82) " " t(11;17)(p11;q11) (84) " (86) (87) " c-myc amplification (88) P.

However in Stage III and IV NB where tumor extends beyond the midline, is metastatic or involves bony lesions the prognosis is poor. It is possible that early stage disease (Stage I,II, IVS) is a distinct entity from late stage (Stage III, IV) disease because their response rate to therapy and their molecular genetic and biologic characteristics are distinct. Of all human tumors neuroblastomas have one of the highest rates of spontaneous tumor regression. This is primarily due to an unusual type of neuroblastoma called Stage IVS that occurs in infants under a year of age who present with widely disseminated disease that typically resolves with minimal therapy.

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